minianalysis version 4.3.1 (synaptosoft inc)
90
Structured Review
synaptosoft inc
minianalysis version 4.3.1
Minianalysis Version 4.3.1, supplied by synaptosoft inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/minianalysis+version+4%2E3%2E1/minianalysis+software/pm32007398-127-6-9
Average 90 stars, based on 1 article reviews
Minianalysis Version 4.3.1, supplied by synaptosoft inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/minianalysis+version+4%2E3%2E1/minianalysis+software/pm32007398-127-6-9
Average 90 stars, based on 1 article reviews
minianalysis version 4.3.1 - by Bioz Stars,
2026-09
90/100 stars
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other:Article Title: Quercetin Enhances Inhibitory Synaptic Inputs and Reduces Excitatory Synaptic Inputs to OFF- and ON-Type Retinal Ganglion Cells in a Chronic Glaucoma Rat Model. Article Snippet: The frequency and amplitude parameters of miniature synaptic currents were analyzed by Article Title: Inspiratory-Activated Airway Vagal Preganglionic Neurones Excited by Thyrotropin-Releasing Hormone via Multiple Mechanisms in Neonatal Rats Article Snippet: Spontaneous or miniature synaptic currents, as well as the ICSs phase-locked to the rapid inward phase of FOCs, were analyzed with Article Title: Corticotropin-releasing hormone modulates airway vagal preganglionic neurons of Sprague-Dawley rats at multiple synaptic sites via activation of its type 1 receptors: Implications for stress-associated airway vagal excitation. Article Snippet: Corticotropin-releasing hormone release is the final common pathway of stress-associated neuroendocrine responses.. This study tested how corticotropin-releasing hormone modulates airway vagal preganglionic neurons.. Airway vagal preganglionic neurons in neonatal rats were retrogradely labeled with fluorescent dye and identified in medullary slices, and their responses to corticotropin-releasing hormone (200 nmol L) were examined using whole-cell patch clamp. Article Title: Moxonidine inhibits excitatory inputs to airway vagal preganglionic neurons via activation of both α 2 -adrenoceptors and imidazoline I1 receptors. Article Snippet: As an imidazoline I1 receptor agonist with very weak binding affinity for 2-adrenoceptors, moxonidine is commonly used in the treatment of hypertension.. Moxonidine also has been implicated to act centrally to reduce airway vagal outflow.. However, it is unknown at which central sites moxonidine acts to affect airway vagal activity, and how moxonidine takes effect at synaptic and receptor levels. Article Title: Asthmatic Augmentation of Airway Vagal Activity Involves Decreased Central Expression and Activity of CD73 in Rats. Article Snippet: The severity of asthma is closely related to the intensity of airway vagal activity; however, it is unclear how airway vagal activity is centrally augmented in asthma.. Here we report that in an asthma model of male Sprague-Dawley rats, the expression and activity of ecto-5'-nucleotidase (CD73) were decreased in airway vagal centers; ATP concentration in cerebral spinal fluid was increased; and the inhibitory and excitatory airway vagal responses to intracisternally injected ATP (5 mol) and CD73 inhibitor AMPCP (5 mol), respectively, were attenuated.. In airway vagal preganglionic neurons (AVPNs) identified in medullary slices of neonatal Sprague-Dawley rats, AMPCP (100 mol·L-1) caused excitatory effects, as are shown in patch-clamp by depolarization, increased neuronal discharge, and facilitated spontaneous excitatory postsynaptic currents (sEPSCs). Article Title: Chronic Intermittent Hypoxia and Hypercapnia Inhibit the Hypothalamic Paraventricular Nucleus Neurotransmission to Parasympathetic Cardiac Neurons in the Brain Stem Article Snippet: The analysis of prolonged responses (>0.5 seconds) was performed using Article Title: Activation of α1-adrenoceptors facilitates excitatory inputs to medullary airway vagal preganglionic neurons. Article Snippet: Dengyun Ge,* Xianxia Yan,* Yuhong Guo, Xingxin Chen, Ruijuan Guan, Yonghua Chen, Dongying Qiu, and Jijiang Wang Department of Neurobiology, School of Basic Medical Sciences, Fudan University, Shanghai, China; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University, Shanghai, China; and Department of Gerontology, Zhongshan Hospital, Fudan University, Shanghai, China |